Non-toxic inhibitor for esterase, lipase and N-formyl-methionine aminopeptidase. Inhibits also carboxypeptidase Y-like exopeptidase.

酯、脂肪和N-甲酰甲硫氨酸氨基肽无毒的抑制剂。也抑制羧肽Y类外肽。

Cutinase is an exteacellular enzyme which catalyses the hydrosis of cutin and belongs to aclass of serine esterases.

角质是一种可以降解植物角质的水解,属于丝氨酸酯。

After absorption, prulifloxacin is metabolised by esterases to ulifloxacin, which inhibits the formation of superhelix, cell division and reproduction by interfering the DNA toposomerase.

为前体药,其性代谢产物碍DNA拓扑异构使细菌DNA无法形成超螺旋,导致细菌细胞无法分裂繁殖。

The results of biochemical analysis indicated that the difference of the activity of carboxylesterase and esterase isozymes in the larval midguts were the main mechanism of this selective toxicity.

生化分析结果进一步指出,昆虫中肠羧酸酯的性及酯同功的差异是这种选择麻醉作用的重要机制。

Non-toxic inhibitor for esterase, lipase and N-formyl-methionine aminopeptidase. Inhibits also carboxypeptidase Y-like exopeptidase.

酯酶、脂肪酶和N-甲酰甲硫氨酸氨基肽酶无毒的抑制剂。也抑制羧肽酶Y类外肽酶。

Cutinase is an exteacellular enzyme which catalyses the hydrosis of cutin and belongs to aclass of serine esterases.

角质酶是一降解植物角质的水解酶,属于丝氨酸酯酶。

After absorption, prulifloxacin is metabolised by esterases to ulifloxacin, which inhibits the formation of superhelix, cell division and reproduction by interfering the DNA toposomerase.

本品为前体药,其性代谢产物通过阻碍DNA拓扑异构酶使菌DNA无法形成超螺旋,菌胞无法分裂繁殖。

The results of biochemical analysis indicated that the difference of the activity of carboxylesterase and esterase isozymes in the larval midguts were the main mechanism of this selective toxicity.

生化分析结果进一步指出,昆虫中肠羧酸酯酶的性及酯酶同功酶的差异是这选择麻醉作用的重要机制。

Non-toxic inhibitor for esterase, lipase and N-formyl-methionine aminopeptidase. Inhibits also carboxypeptidase Y-like exopeptidase.

酯酶、脂肪酶和N-甲酰甲硫氨酸氨基肽酶无毒的抑制剂。也抑制羧肽酶Y类外肽酶。

Cutinase is an exteacellular enzyme which catalyses the hydrosis of cutin and belongs to aclass of serine esterases.

角质酶是以降解植物角质的水解酶,属于丝氨酸酯酶。

After absorption, prulifloxacin is metabolised by esterases to ulifloxacin, which inhibits the formation of superhelix, cell division and reproduction by interfering the DNA toposomerase.

本品为前体药,其性代谢产物通过阻碍DNA拓扑异构酶使DNA无法形成超螺旋,导胞无法分裂繁殖。

The results of biochemical analysis indicated that the difference of the activity of carboxylesterase and esterase isozymes in the larval midguts were the main mechanism of this selective toxicity.

生化分析结果进步指出,昆虫中肠羧酸酯酶的性及酯酶同功酶的差异是这选择麻醉作用的重要机制。

Non-toxic inhibitor for esterase, lipase and N-formyl-methionine aminopeptidase. Inhibits also carboxypeptidase Y-like exopeptidase.

酯、脂肪和N-氨酸氨基肽无毒抑制剂。也抑制羧肽Y类外肽。

Cutinase is an exteacellular enzyme which catalyses the hydrosis of cutin and belongs to aclass of serine esterases.

角质是一种可以降解植物角质水解,属于丝氨酸酯。

After absorption, prulifloxacin is metabolised by esterases to ulifloxacin, which inhibits the formation of superhelix, cell division and reproduction by interfering the DNA toposomerase.

本品为前体药,其性代谢产物通过阻碍DNA拓扑异构使细菌DNA无法形成超螺旋,导致细菌细胞无法分裂繁殖。

The results of biochemical analysis indicated that the difference of the activity of carboxylesterase and esterase isozymes in the larval midguts were the main mechanism of this selective toxicity.

生化分析结果进一步指出,昆虫中肠羧酸酯性及酯同功差异是这种选择麻醉作用重要机制。

Non-toxic inhibitor for esterase, lipase and N-formyl-methionine aminopeptidase. Inhibits also carboxypeptidase Y-like exopeptidase.

酯酶、脂肪酶和N-甲酰甲硫氨酸氨基肽酶无毒的抑制剂。也抑制羧肽酶Y类外肽酶。

Cutinase is an exteacellular enzyme which catalyses the hydrosis of cutin and belongs to aclass of serine esterases.

角质酶是一种可以降解植角质的水解酶,属于丝氨酸酯酶。

After absorption, prulifloxacin is metabolised by esterases to ulifloxacin, which inhibits the formation of superhelix, cell division and reproduction by interfering the DNA toposomerase.

本品药,其性代通过阻碍DNA拓扑异构酶使细菌DNA无法形成超螺旋,导致细菌细胞无法分裂繁殖。

The results of biochemical analysis indicated that the difference of the activity of carboxylesterase and esterase isozymes in the larval midguts were the main mechanism of this selective toxicity.

生化分析结果进一步指出,昆虫中肠羧酸酯酶的性及酯酶同功酶的差异是这种选择麻醉作用的重要机制。

Non-toxic inhibitor for esterase, lipase and N-formyl-methionine aminopeptidase. Inhibits also carboxypeptidase Y-like exopeptidase.

酯酶、脂肪酶和N-甲酰甲硫氨酸氨基肽酶无毒的抑制剂。也抑制羧肽酶Y类外肽酶。

Cutinase is an exteacellular enzyme which catalyses the hydrosis of cutin and belongs to aclass of serine esterases.

酶是一种可以降解植的水解酶,属于丝氨酸酯酶。

After absorption, prulifloxacin is metabolised by esterases to ulifloxacin, which inhibits the formation of superhelix, cell division and reproduction by interfering the DNA toposomerase.

本品为前体药,其性代谢产通过阻碍DNA拓扑异构酶使细菌DNA无法超螺旋,导致细菌细胞无法分裂繁殖。

The results of biochemical analysis indicated that the difference of the activity of carboxylesterase and esterase isozymes in the larval midguts were the main mechanism of this selective toxicity.

生化分析结果进一步指出,昆虫中肠羧酸酯酶的性及酯酶同功酶的差异是这种选择麻醉作用的重要机制。

Non-toxic inhibitor for esterase, lipase and N-formyl-methionine aminopeptidase. Inhibits also carboxypeptidase Y-like exopeptidase.

酯、脂肪和N-甲酰甲硫氨酸氨基肽无毒的抑制剂。也抑制羧肽Y类外肽。

Cutinase is an exteacellular enzyme which catalyses the hydrosis of cutin and belongs to aclass of serine esterases.

角质是一种可以降解植物角质的水解,属于丝氨酸酯。

After absorption, prulifloxacin is metabolised by esterases to ulifloxacin, which inhibits the formation of superhelix, cell division and reproduction by interfering the DNA toposomerase.

为前体药,其性代谢产物碍DNA拓扑异构使细菌DNA无法形成超螺旋,导致细菌细胞无法分裂繁殖。

The results of biochemical analysis indicated that the difference of the activity of carboxylesterase and esterase isozymes in the larval midguts were the main mechanism of this selective toxicity.

生化分析结果进一步指出,昆虫中肠羧酸酯的性及酯同功的差异是这种选择麻醉作用的重要机制。

Non-toxic inhibitor for esterase, lipase and N-formyl-methionine aminopeptidase. Inhibits also carboxypeptidase Y-like exopeptidase.

酶、脂肪酶和N-甲酰甲硫氨酸氨基肽酶无毒的抑制剂。也抑制羧肽酶Y类外肽酶。

Cutinase is an exteacellular enzyme which catalyses the hydrosis of cutin and belongs to aclass of serine esterases.

角质酶是一种可以降解植物角质的水解酶,属于丝氨酸酶。

After absorption, prulifloxacin is metabolised by esterases to ulifloxacin, which inhibits the formation of superhelix, cell division and reproduction by interfering the DNA toposomerase.

本品为前体药,其性代谢产物通过阻碍DNA拓扑异构酶使细菌DNA无法形成超螺旋,导致细菌细胞无法分裂繁殖。

The results of biochemical analysis indicated that the difference of the activity of carboxylesterase and esterase isozymes in the larval midguts were the main mechanism of this selective toxicity.

生化分析结果进一步指出,昆虫中肠羧酸酶的性及酶同功酶的差异是这种醉作用的重要机制。

Non-toxic inhibitor for esterase, lipase and N-formyl-methionine aminopeptidase. Inhibits also carboxypeptidase Y-like exopeptidase.

、和N-甲酰甲硫氨酸氨基肽无毒的抑制剂。也抑制羧肽Y类外肽。

Cutinase is an exteacellular enzyme which catalyses the hydrosis of cutin and belongs to aclass of serine esterases.

角质是一种可以降解植物角质的水解,属于丝氨酸。

After absorption, prulifloxacin is metabolised by esterases to ulifloxacin, which inhibits the formation of superhelix, cell division and reproduction by interfering the DNA toposomerase.

本品为前体药,其性代谢产物通过阻碍DNA拓扑异构使细菌DNA无法形成超螺旋,导致细菌细胞无法分裂繁殖。

The results of biochemical analysis indicated that the difference of the activity of carboxylesterase and esterase isozymes in the larval midguts were the main mechanism of this selective toxicity.

生化分析结果进一步指出,昆虫中肠羧酸的性及功的差异是这种选择麻醉作用的重要机制。

Non-toxic inhibitor for esterase, lipase and N-formyl-methionine aminopeptidase. Inhibits also carboxypeptidase Y-like exopeptidase.

酯酶、脂肪酶和N-甲酰甲硫氨酸氨基肽酶无毒抑制剂。也抑制羧肽酶Y类外肽酶。

Cutinase is an exteacellular enzyme which catalyses the hydrosis of cutin and belongs to aclass of serine esterases.

角质酶是一种可以降解植物角质水解酶,属于丝氨酸酯酶。

After absorption, prulifloxacin is metabolised by esterases to ulifloxacin, which inhibits the formation of superhelix, cell division and reproduction by interfering the DNA toposomerase.

本品为前体药,其性代谢产物通过阻碍DNA拓扑异构酶使细菌DNA无法形成超螺旋,导致细菌细胞无法分裂繁殖。

The results of biochemical analysis indicated that the difference of the activity of carboxylesterase and esterase isozymes in the larval midguts were the main mechanism of this selective toxicity.

生化分析结果进一步指出,昆虫中肠羧酸酯酶性及酯酶同功酶差异是这种选择麻醉重要机制。