Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导致夜盲、毛囊周角化度、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化度,呈乳头瘤样增生,颗粒层增厚,真皮乳头见大量增生扩张的毛细血管,管腔内含红细胞,血管周围淋巴细胞浸润。

Histopathology showed moderate hyperkeratosis and rete ridge elongation with the epidermal thinning above the papillar layer and a few melanophages in the dermis.

腋下、腹股沟、乳房下色素沉着15年,无显自觉症状。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导致夜盲、毛囊周角化、笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化,呈乳头瘤样增生,颗粒层增厚,真皮乳头见大量增生扩张的毛细,腔内含红细胞,周围淋巴细胞浸润。

Histopathology showed moderate hyperkeratosis and rete ridge elongation with the epidermal thinning above the papillar layer and a few melanophages in the dermis.

腋下、腹股沟、乳房下色素沉着15年,无明显自觉症状。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导致夜盲、毛囊周角化度、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化度,呈乳头瘤样增生,颗粒层增厚,真皮乳头见大量增生扩张的毛细血管,管腔内含红细胞,血管周围淋巴细胞浸润。

Histopathology showed moderate hyperkeratosis and rete ridge elongation with the epidermal thinning above the papillar layer and a few melanophages in the dermis.

腋、沟、乳房色素沉着15年,无明显自觉症状。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导致夜盲、毛囊度、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮度,呈乳头瘤样增生,颗粒层增厚,真皮乳头见大量增生扩张的毛细血管,管腔细胞,血管围淋巴细胞浸润。

Histopathology showed moderate hyperkeratosis and rete ridge elongation with the epidermal thinning above the papillar layer and a few melanophages in the dermis.

腋下、腹股沟、乳房下色素沉着15年,无明显自觉症状。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导致夜盲、毛囊周角化、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化,呈乳头瘤样增生,颗粒层增厚,真皮乳头见大量增生扩张的毛细血管,管腔内含红细胞,血管周围淋巴细胞浸润。

Histopathology showed moderate hyperkeratosis and rete ridge elongation with the epidermal thinning above the papillar layer and a few melanophages in the dermis.

腋下、腹股沟、乳房下色素沉着15年,无明状。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导致夜盲、角化度、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化度,呈乳头瘤样增生,颗粒层增厚,真皮乳头见大量增生扩张的血管,管腔内含,血管围淋巴浸润。

Histopathology showed moderate hyperkeratosis and rete ridge elongation with the epidermal thinning above the papillar layer and a few melanophages in the dermis.

腋下、腹股沟、乳房下色素沉着15年,无明显自觉症状。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导致夜盲、角化度、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化度,呈乳头瘤样增生,颗粒层增厚,真皮乳头见大量增生扩张的血管,管腔内含,血管围淋巴浸润。

Histopathology showed moderate hyperkeratosis and rete ridge elongation with the epidermal thinning above the papillar layer and a few melanophages in the dermis.

腋下、腹股沟、乳房下色素沉着15年,无明显自觉症状。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导、毛囊角化度、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化度,呈乳头瘤样增生,颗粒层增厚,真皮乳头见大量增生扩张的毛细,腔内含红细胞,围淋巴细胞浸润。

Histopathology showed moderate hyperkeratosis and rete ridge elongation with the epidermal thinning above the papillar layer and a few melanophages in the dermis.

腋下、腹股沟、乳房下色素沉着15年,无明显自觉症状。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导致夜盲、毛囊周角化度、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化度,呈头瘤样增生,颗粒层增厚,真皮头见大量增生扩张的毛细血管,管腔内含红细胞,血管周围淋巴细胞浸润。

Histopathology showed moderate hyperkeratosis and rete ridge elongation with the epidermal thinning above the papillar layer and a few melanophages in the dermis.

腋下、腹、房下色素沉着15年,无明显自觉症状。