Abstract The effects of neurotransmitters and receptor antagonists on HFO in phrenic discharge were observed in 65 unanaesthetized, vagotomized, paralyzed rabbits.
MB leads little changes of hemodynamics due to inhibit cardiac sympathicotonia and pneumogastric nerve simultaneity, but TEB brings the evident changes due to only inhibit pneumogastric nerve.
That sensory input gets relayed to the hypothalamus, which stimulates the medulla oblongata, which then taps the parasympathetic fibers in the vagus nerve.